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X-WR-CALDESC:Events for UW Neurobiology & Biophysics
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DTSTART;TZID=America/Los_Angeles:20251103T180000
DTEND;TZID=America/Los_Angeles:20251103T210000
DTSTAMP:20260424T213945
CREATED:20251020T185257Z
LAST-MODIFIED:20251020T185441Z
UID:10000120-1762192800-1762203600@nbio.uw.edu
SUMMARY:Art Neureau
DESCRIPTION:Art Neureau is a neuroscience-themed art show hosted by UW Graduate Program in Neuroscience and the Eberhard Fetz Art Fund. \n 
URL:https://nbio.uw.edu/event/art-neureau/
LOCATION:Fremont Abbey Arts Center\, 4272 Fremont Ave N\, Seattle\, 98103\, United States
CATEGORIES:Single day event
ORGANIZER;CN="Graduate Program Neuroscience":MAILTO:neurogrd@uw.edu
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BEGIN:VEVENT
DTSTART;TZID=America/Los_Angeles:20251106T000000
DTEND;TZID=America/Los_Angeles:20251106T235959
DTSTAMP:20260424T213945
CREATED:20251105T162607Z
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UID:10000122-1762387200-1762473599@nbio.uw.edu
SUMMARY:Breaking Barriers in Science Symposium
DESCRIPTION:Please join the Department of Neurology and the Department of Neurobiology & Biophysics for Breaking Barriers in Brain Science: A Neuroscience Symposium for UW Medicine. \nWe welcome University of Washington researchers from neurology\, neurosurgery\, neurobiology\, bioengineering\, rehabilitation\, and related fields to attend. This symposium offers a valuable opportunity to share ideas\, build new partnerships\, and accelerate cross-departmental discovery.
URL:https://nbio.uw.edu/event/breaking-barriers-in-science-symposium/
CATEGORIES:Single day event
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DTSTART;TZID=America/Los_Angeles:20251113T093000
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CREATED:20251027T221535Z
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UID:10000121-1763026200-1763029800@nbio.uw.edu
SUMMARY:NBIO Presents: Dana Miller\, PhD (University of Washington)
DESCRIPTION:“Genetic and epigenetic regulation of H2S homeostasis in C. elegans”\nHydrogen sulfide (H2S) was once known primarily as a toxic gas with the smell of rotten eggs. However\, H2S also has many roles as a cellular messenger that can promote survival in stressful conditions. Both endogenously produced and exogenously supplied H2S protect against cellular damage and death associated with ischemia/reperfusion injury in mammals. We use C. elegans to explore mechanisms of beneficial and toxic effects of H2S in animals. We have shown that C. elegans grown in 50 ppm H2S are long-lived and resistant to hypoxia-induced disruption of protein homeostasis\, and that the early transcriptional response to H2S requires the C. elegans orthologue of the hypoxia-inducible transcription factor\, hif-1. HIF-1 promotes survival in H2S\, at least in part\, by upregulating expression of sqrd-1\, which encodes the sulfide-quinone oxidoreductase. SQRD-1 catalyzes the first step in the mitochondrial oxidation of H2S\, the only known cellular pathway for H2S catabolism. We have found that exposure to H2S activates an epigenetic response that forms a transcriptional memory that enables the animals to survive subsequent exposure to otherwise lethal H2S. We have also identified a protein\, RHY-1\, that can promote H2S tolerance even in the absence of the hif-1-mediated transcriptional response or the sqrd-1-mediated H2S oxidation. RHY-1 is an integral- membrane ER protein with predicted acyltransferase (ACYL3) activity. The ACYL3 family is large and conserved across species from bacteria to primates\, but the function of these enzymes is largely unstudied. We identified a novel methyltransferase\, RIPS-1\, that is specifically required for RHY-1 to promote survival in H2S using biotinylation by antibody recognition (BAR). Our preliminary genetic evidence suggests that cholinergic signaling is required for the protective effects of RHY-1 expression in H2S. \nIn Health Sciences G-328 and on Zoom
URL:https://nbio.uw.edu/event/nbio-presents-dana-miller-phd-university-of-washington/
LOCATION:Health Sciences G-328
CATEGORIES:Seminar
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ORGANIZER;CN="UW NBIO":MAILTO:nbio@uw.edu
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DTSTART;TZID=America/Los_Angeles:20251120T093000
DTEND;TZID=America/Los_Angeles:20251120T103000
DTSTAMP:20260424T213945
CREATED:20250905T214302Z
LAST-MODIFIED:20260403T163807Z
UID:10000088-1763631000-1763634600@nbio.uw.edu
SUMMARY:NBIO Presents: Amy Arnsten\, PhD (Yale University)
DESCRIPTION:“Dynamic Network Connectivity: The unusual molecular regulation of dorsolateral prefrontal cortex confers vulnerability to cognitive disorders”\nThe dorsolateral prefrontal cortex (dlPFC) subserves high order cognitive functions such as working memory and abstract thought and the top-down control of attention\, action and emotion. However\, the dlPFC is remarkably fragile\, impaired by fatigue\, stress\, aging and inflammation\, and is dysfunctional in most cognitive disorders. Arnsten’s lab has discovered the unusual molecular mechanisms that govern these circuits\, which are essential for generating and sustaining representations in working memory and for coordinating cognitive state with arousal state. However\, these same mechanisms can lead to atrophy and tau pathology when dysregulated by stress and/or inflammation. Knowledge of these mechanisms has led to treatments for cognitive disorders in patients. \n Health Sciences G328 and Zoom
URL:https://nbio.uw.edu/event/nbio-presents-amy-arnsten-yale-university/
LOCATION:Health Sciences G-328
CATEGORIES:Seminar
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ORGANIZER;CN="UW NBIO":MAILTO:nbio@uw.edu
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